15 :30 Welcoming
16:00 to 17:30 : Periimplant diseases (online presentation)
18:00 to 19:00 : Postgraduate program in Periodontology ULiège - EFP thesis defense : Yaman AL TAEP: “Influence of periodontitis and periodontal treatment on Alzheimer’s disease progression"
EFP examiner : Prof. Jose Nart
20:00 : Proclamation & Cocktail
Biography : Prof. Dr. Jose Nart
- Doctor in Dental Surgery (DDS)
Universitat Internacional de Catalunya (UIC-Barcelona), Spain - PhD in Dentistry
Universitat Internacional de Catalunya (UIC-Barcelona), Spain - Certificate in Periodontology and Implants
Tufts University, Boston, USA. - Diplomate, American Board of Periodontology and Implant Dentistry
- Professor. Chairman and Program Director, Department of Periodontology, UIC-Barcelona, Spain. European Federation of Periodontology (EFP) accredited program
- Vice-President, Spanish Society of Periodontology and Osseointegration (SEPA)
- Vice-Dean of Research, Universitat Internacional de Catalunya, Barcelona, Spain 2017-19.
- Expert Council Committee member, Osteology Foundation
- Chairman, 49th Annual Meeting Barcelona 2015, Spanish Society of Periodontology and Osseointegration (SEPA)
- International Team of Implantology (ITI) Fellow
- Reviewer for Journal of Periodontology, Journal of Clinical Periodontology, Clinical Oral Implant Research, Clinical Implant Dentistry and Related Research, Clinical Oral Investigations
- National and International lecturer
- International high-impact factor scientific publications (+70 JCR)
Influence of periodontitis and periodontal treatment on Alzheimer’s disease progression
Periodontitis causes systemic inflammation through the release of periodontal pathogens and byproducts in the blood stream. As chronic intracerebral inflammation is known to trigger the progression of Alzheimer’s Disease (AD), researchers suggested potential associations between the two disorders. Indeed, specific periodontal pathogens can alter the brain neuroinflammation, influence the amyloid and the tau protein metabolism ultimately contributing to the destruction of neurons, thus resulting in an amplified progression of AD. Therefore we hypothesized that reduction of the systemic inflammation by providing periodontal treatment in AD patients may positively affect AD features.
Therefore, during this thesis defense, the physiopathological mechanisms linking the two disorders in pre-clinical studies will be explored and discussed. Based on this information, the periodontal treatment strategy in AD patients will be presented through a randomized clinical trial.
